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	<title>PhysioDigest - an educational resource for the musculoskeletal rehabilitation community &#187; TMJ</title>
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		<title>Headache, Orofacial Pain and Bruxism</title>
		<link>http://www.physiodigest.com/4995/headache-orofacial-pain-and-bruxism/?utm_source=rss&#038;utm_medium=rss&#038;utm_campaign=headache-orofacial-pain-and-bruxism</link>
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		<pubDate>Wed, 16 Dec 2009 13:31:04 +0000</pubDate>
		<dc:creator>David Fitzgerald</dc:creator>
		
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		<description><![CDATA[The International Headache Society originally classified 126 different types of headaches and orofacial pain.  In primary care 3 sub-classifications of headaches allow useful patient categorisation. Migraine Tension type headache Cervicogenic headache. Orofacial pain and Bruxism often compound the diagnosis of headache as there maybe overlapping pathophysiology. Exclusion of “red flags” is critical. Headache “Red Flags” [...]]]></description>
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<p>The International Headache Society originally classified 126 different types of headaches and orofacial pain.  In primary care 3 sub-classifications of headaches allow useful patient categorisation.</p>
<p>Migraine</p>
<p>Tension type headache</p>
<p>Cervicogenic headache.</p>
<p>Orofacial pain and Bruxism often compound the diagnosis of headache as there maybe overlapping pathophysiology. Exclusion of “red flags” is critical.</p>
<h2><strong>Headache “Red Flags”</strong></h2>
<p><strong> </strong></p>
<p>1)      Instantaneous headache</p>
<p>2)      Sub-acute headache in patients over 55</p>
<p>3)      Neurological signs</p>
<p>4)      Fever</p>
<p>5)      Neck stiffness</p>
<p>6)      Headache worse on wakening</p>
<p>7)      Recent head trauma.</p>
<p>Having excluded the red flag categorisations you are then left with the three defining categories as above of migraine, tension type headache and cervicogenic headache.</p>
<h2>Migraine</h2>
<p>Symptoms &#8230;..</p>
<p>Fequently intense</p>
<p>more likely to be unilateral</p>
<p>Pounding</p>
<p>Nausea</p>
<p>Photophobia</p>
<p>Phonophobia</p>
<p>Helped by sleep.</p>
<p>Typically occurring episodes lasting between 4-72 hours.</p>
<p>True migraine can be managed by the use of pharmacological agents such as:</p>
<p>Ergotamine</p>
<p>Triptan</p>
<p>Simple analgesics</p>
<p>Antiemetic medications.</p>
<p>These are obviously issues for a prescribing physician or neurologist.  The physiotherapist needs to be aware of these therapeutic modalities as often the patient will present with neck pain which is secondary to a primary vascular cause of migraine.  Detailed history of the migraine pattern in terms of precipitating factors which maybe well known food substances e.g. chocolate, red wine, dairy products or less frequently dehydration, fatigue, exercise, physical loading, ergonomics and musculoskeletal factors.  While these patients may have true vascular migraine, mechanical factors amenable to physiotherapy may also be relevant triggers and thus are legitimate targets for treatment.</p>
<h2>Tension-Type Headache</h2>
<p>Very often a chronic migraineur develops a pattern, persistent tension type headache as part of an ongoing symptom pattern punctuated by episodes of an acute vascular migraine.  The ongoing tension headache is often more debilitating and functionally impairing and harder to manage with pharmacological strategies.  Typically described as:</p>
<p>Dull pressure</p>
<p>Band like pain radiating from the forehead to the occiput</p>
<p>Often involving the neck muscles.</p>
<p>Pathogenesis is unclear at this point.  In association with persistent muscle tension the obvious therapeutic strategies of optimising muscle control, improving deep neck flexor function and optimising scapula control are all part of the routine testing protocols which should be addresses if clinically demonstrated.  For patients who do not display these deficits there maybe a role for general relaxation or specific biofeedback training. The commonest clinical challenge is to prioritise from a multitude of co-existing findings.</p>
<h2>Cervicogenic Headache</h2>
<p>This is frequently under recognised in primary care masquerading under a diagnosis of migraine.  Typically these patients describe symptoms, which last longer than an acute migraine episode of 4-72 hours with symptoms often lasting days to weeks. Sleep does not usually influence symptoms and the pain intensity is usually of less severity that an acute migraine attack.  There may be some associated sensations of aura mimicking a migraine. These can sometimes be reproduced by cervical provocation testing using manual techniques.  Pain is usually localised on one side and frequently periorbital distribution but can also involve the maxillary and mandibular regions. Co-existing TMJ pathology may compound the diagnosis.</p>
<h2>TMJ Related Headache</h2>
<p>The pain referral pattern from the TMJ may involve the temporal and frontal regions and the myofascial referral zones involve the head, face and neck.  The primary issue with TMJ related pain is to establish whether it is driven by Dental issues relating to occlusion and bite mechanics, which require specific intervention from those specialties.</p>
<p>Alternatively, Bruxism (habitual grinding) &#8211; both nocturnal and daily are often significant perpetuating factors in the symptom pattern.  Treatments that target the TMJ involve specific joint mobilisation, improving general ranges of motion, post-isometric  relaxation, general jaw mobilisation and specific myofascial trigger point therapy using either palpatory of dry needling techniques.  Dental occlusion splints at night are also typically part of the management strategy.</p>
<p>From a physiotherapy perspective it is common to observe  a temporary reduction in clinical signs by treating symptomatic articular / myofascial components but the effects of treatment are relatively short lived.  In these situations the challenge is then to explore symptom drivers.  The use of kinesio tape is beneficial for evaluating the role of postural alignment as often there is a dual ergonomic and stress / tension component related to occupational factors as part of the symptomatology.  These are undoubtedly challenging patients to treat and certainly require multi disciplinary collaborative input in order to optimise management.</p>
<p>Enjoy the clinical challenge</p>
<p>David</p>
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Post tags: <a href="http://www.physiodigest.com/tag/cervicogenic-headache/" rel="tag">cervicogenic headache</a>, <a href="http://www.physiodigest.com/tag/headache/" rel="tag">Headache</a>, <a href="http://www.physiodigest.com/tag/headache-back-of-head/" rel="tag">headache back of head</a>, <a href="http://www.physiodigest.com/tag/migraine/" rel="tag">migraine</a>, <a href="http://www.physiodigest.com/tag/orofacial-pain/" rel="tag">orofacial pain</a>, <a href="http://www.physiodigest.com/tag/tension-headache/" rel="tag">tension headache</a>, <a href="http://www.physiodigest.com/tag/tmj/" rel="tag">TMJ</a>, <a href="http://www.physiodigest.com/tag/tmj-disorder/" rel="tag">TMJ disorder</a>, <a href="http://www.physiodigest.com/tag/tmj-myofascial-pain/" rel="tag">tmj myofascial pain</a>, <a href="http://www.physiodigest.com/tag/tmj-symptoms/" rel="tag">TMJ symptoms</a>, <a href="http://www.physiodigest.com/tag/tmj-syndrome/" rel="tag">TMJ syndrome</a>, <a href="http://www.physiodigest.com/tag/types-of-headaches/" rel="tag">types of headaches</a>, <a href="http://www.physiodigest.com/tag/types-of-migraine-headache/" rel="tag">types of migraine headache</a><br/>
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		<title>TMJ &amp; Facial Pain</title>
		<link>http://www.physiodigest.com/875/tmj-facial-pain/?utm_source=rss&#038;utm_medium=rss&#038;utm_campaign=tmj-facial-pain</link>
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		<pubDate>Fri, 28 Aug 2009 06:00:45 +0000</pubDate>
		<dc:creator>David Fitzgerald</dc:creator>
		
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		<description><![CDATA[TMJ and facial pain is another one of those challenging conditions where the pathology is not easy to define and several specialities can be involved in co-ordinating patient management.  These may include dentists, pain management specialists, ENT, neurology, physiotherapy and the whole myriad of allied health professionals.  It is often difficult to quantify whether the [...]]]></description>
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<p>TMJ and facial pain is another one of those challenging conditions where the pathology is not easy to define and several specialities can be involved in co-ordinating patient management.  These may include dentists, pain management specialists, ENT, neurology, physiotherapy and the whole myriad of allied health professionals.  It is often difficult to quantify whether the facial symptoms are primary or secondary in nature even when a specific mechanical history is reported.  One of the reasons for this is that often a traumatic mechanism involving the head and neck can simultaneously irritate multiple structures and therefore doesn&#8217;t narrow down the examination process.  Similarly mechanisms such as whiplash which do not directly involve the jaw as a point of impact also appear to be capable of inducing secondary irritation either by way of reflex changes or as a consequence of the inertia from violent head and upper thorax motion.</p>
<div class="title-h1">TMJ Symptoms</div>
<p>Specific symptom reports such as clicking, locking, opening restriction, pain on bite, fatigue on talking and slowness of speech are all frequent symptom reports from patients with primary TMJ problems.  There may well be some localised tenderness along the joint line also.</p>
<p>When patients describe overt locking and clicking there are a number of possibilities to be considered.</p>
<div class="title-h1">Primary TMJ Pain</div>
<p>1)      Intra-articular disc pathology &#8211; This is usually a clinical diagnosis which requires arthroscopic confirmation and management.</p>
<p>2)      Intra-articular impingement secondary to capsular restriction &#8211; Patients in this grouping display restricted motion of the TMJ sometimes in a global pattern and sometimes direction specific.  The primary treatment strategy in this patient group is to improve joint mobility by re-evaluating the intra-articular pain pattern post-intervention.</p>
<p>3)      TMJ instability &#8211; This is a most challenging group where the structural constraints have become either torn or chronically elongated thus compromising stability in the region.  Patients in this category are often the poor responders to treatment or those who require on going maintenance as part of their management strategy.</p>
<div class="title-h1">Secondary TMJ Pain</div>
<p>Secondary TMJ Pain &#8211; Myofascial pain radiating to the face, head and jaw is extremely common.  In the presence of a post whiplash history there will often be co-existing cervicogenic signs, headaches and even neuropathic symptoms in the head and face as part of the general symptom pattern.  In clear-cut cervicogenic myofascial referral the symptoms can be reproduced by palpation of trigger points in;</p>
<p>Trapizus</p>
<p>Sternocleidomastoid</p>
<p>Scalene</p>
<p>Scapular musculature.</p>
<p>Specific evaluation of the face and jaw with palpatory techniques does usually not yield sufficient information but diagnostic probing using dry needling is certainly worthwhile. Combinations of manual therapy specifically to the joint condyles together with hold- relax procedures in the direction of motion restriction are all part of the tools normally used to manage this type of condition.  The therapist should be aware that intra-oral techniques are usually required so appropriate gloving and preparatory procedures are necessary.</p>
<div class="title-h1">Bruxism</div>
<p>A common clinical challenge we face is patients who have regular sleep disturbance following a traumatic neck injury.  This cycle of sleep disturbance if sustained for any period induces fatigue with subsequent diffuse increases in muscle tension.  When this cycle involves into a pattern of habitual teeth grinding, known as &#8216;Bruxism&#8217; this seriously compounds the clinical management.  It is particularly important to identify in persistent head and facial pain and the clinician is looking for a pattern of temporary response to localised modalities but recurrence of symptoms on an on going basis.  Typically these patients don&#8217;t show gross range of motion restrictions but do exhibit diffuse sensitivity on palpatory testing.</p>
<div class="title-h1">Treating Bruxism</div>
<p>The strategies used to control Bruxism may involve relaxation, dental splinting, and evaluation of bite mechanics as well as looking at other muscular skeletal and postural drivers to potential persistence of muscle hyperactivity.  Undoubtedly an interesting and challenging caseload and one that often involves collarabation of health care professionals &#8211; true multi-disciplinary pain management.</p>
<p>Enjoy the clinical challenge.</p>
<p>David.</p>
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Post tags: <a href="http://www.physiodigest.com/tag/cervicogenic/" rel="tag">cervicogenic</a>, <a href="http://www.physiodigest.com/tag/ent/" rel="tag">ENT</a>, <a href="http://www.physiodigest.com/tag/facial-pain/" rel="tag">Facial Pain</a>, <a href="http://www.physiodigest.com/tag/fatigue-on-talking/" rel="tag">fatigue on talking</a>, <a href="http://www.physiodigest.com/tag/myofascial-pain/" rel="tag">Myofascial pain</a>, <a href="http://www.physiodigest.com/tag/neurology/" rel="tag">neurology</a>, <a href="http://www.physiodigest.com/tag/opening-restriction/" rel="tag">opening restriction</a>, <a href="http://www.physiodigest.com/tag/pain-on-bite/" rel="tag">pain on bite</a>, <a href="http://www.physiodigest.com/tag/physiotherapy/" rel="tag">physiotherapy</a>, <a href="http://www.physiodigest.com/tag/rapizus-sternocleidomastoid/" rel="tag">rapizus  Sternocleidomastoid</a>, <a href="http://www.physiodigest.com/tag/scalene/" rel="tag">Scalene</a>, <a href="http://www.physiodigest.com/tag/scapular-musculaturebruxism/" rel="tag">Scapular musculature.bruxism</a>, <a href="http://www.physiodigest.com/tag/slowness-of-speech/" rel="tag">slowness of speech</a>, <a href="http://www.physiodigest.com/tag/tmj/" rel="tag">TMJ</a>, <a href="http://www.physiodigest.com/tag/tmj-clicking/" rel="tag">TMJ clicking</a><br/>
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		<title>Headache</title>
		<link>http://www.physiodigest.com/582/headache-post-test/?utm_source=rss&#038;utm_medium=rss&#038;utm_campaign=headache-post-test</link>
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		<pubDate>Wed, 01 Jul 2009 12:14:14 +0000</pubDate>
		<dc:creator>David Fitzgerald</dc:creator>
		
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		<description><![CDATA[Differentiation of the pathogenesis of headaches is a complex clinical challenge. The International Headache Society have classified 126 types of headache on the basis of: symptoms behavioural characteristics regional distribution temporal characteristics responsiveness to pharmacological intervention. Many patients are diagnosed as Migraine sufferers but  do not fit the classical &#8221; migraine with aura&#8221; classification and [...]]]></description>
			<content:encoded><![CDATA[<p>Differentiation of the pathogenesis of headaches is a complex clinical challenge. The International Headache Society have classified 126 types of headache on the basis of:</p>
<p>symptoms</p>
<p>behavioural characteristics</p>
<p>regional distribution</p>
<p>temporal characteristics</p>
<p>responsiveness to pharmacological intervention.</p>
<p>Many patients are diagnosed as Migraine sufferers but  do not fit the classical &#8221; migraine with aura&#8221; classification and are then classified as &#8220;atypical migraine without aura&#8221;.</p>
<p>While many of these patients can identify specific triggers (alcohol, dairy products, chemical irritants, ambient lighting, fatigue and dehydration many are unable to determine triggers.</p>
<p>Unfortunately, many chronic migrainuers develop &#8220;Tension type headache&#8221; or alternatively &#8220;chronic daily headache&#8221;</p>
<p>Physiotherapists dealing with this type of caseload commonly find a multitude of clinical signs in the cervical &amp; thoracic spine together with TMJ dysfunction.</p>
<p>The clinical challenge is to determine the relevance of co-existing physical signs and the prioritisation of legitimate targets to treat.</p>
<p>The pain producing structures may be&#8230;.<br />
Articular</p>
<p>Myofascial</p>
<p>Neural</p>
<p>Common mechanisms may precipitate sensitisation of these structures</p>
<p>Such as</p>
<p>postural adaptation</p>
<p>occupational factors</p>
<p>ergonomics</p>
<p>previous musculoskeletal history</p>
<p>and the clinician must prioritise the primary target tissue.</p>
<p>Often this is influenced by clinician bias as to whether they utilise a manual therapy approach, a general exercise regime, a specific muscle imbalance protocol, myofascial techniques or dry needling.</p>
<p>The direction for future research into cervicogenic headaches needs to look at the testing and interpretation of musculoskeletal clinical signs which are frequently associated with headaches in order to establish clinical prediction rules and a clinical efficacy protocol for the management of these challenging patients.</p>
<p>Share your experiences with fellow clinicians by adding your comments below.</p>
<p>Enjoy the clinical challenge</p>
<p>David</p>
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